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How can the FQ's cause different disease processes?

This happens because of the complex interaction that exists between the hundreds of genes and cells that work together to keep the body’s metabolic machinery running smoothly. Just like many mitochondrial diseases that produce varying clinical manifestations from identical mtDNA mutations, FQ's, who influence the mitochondria extensively, may not produce identical clinical manifestations. Therefore, two people can take the same FQ, the same dose, and experience an adverse event but may not look the same clinically, which complicates diagnosis.
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"I wish every time a provider wrote a prescription for a quinolone they were forced to also write on the prescription the type of infection being treated, if there were confirmed microbial cultures growing pseudomonas, and if the patient has drug allergies. That way, the provider will be forced to put more thought into prescribing cipro. Also, the pharmacist can double check if the situation is complicated and serious, or simple and easily cured with a safer antibiotic." ~ Gunda Siska, PharmD

Another good idea to get doctor's thinking!

www.pharmacytimes.com/contributor/gunda-siska-pharmd/2017/08/putting-a-face-on-black-box-warnings
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Do the FQ’s always do damage to DNA for their adverse effects to be experienced long term or chronic?

No. While it is true that they can damage DNA (the subject for another post), their long term negative effects can also be experienced via epigenetic changes. With epigenetic changes, the DNA is not damaged per say, but instead genes which have the capability to cause unwanted health problems are either ‘turned on’ or ‘turned off’ in the right combination.

There is a myth that all mitochondrial diseases are maternally inherited. As a matter of fact most secondary mitochondrial disorders (those experienced by adults later on in life such as floxies), are the result of another process, such as a toxin (FQ) exposure. Many floxies who are chronically effected long term are good examples for adult mitochondrial dysfunction. According to respected mitochondrial disease expert Bruce Cohen M.D. of the Akron Children’s Hospital, formerly of the Cleveland Clinic, these secondary mitochondrial disorders are the result of nuclear DNA that has been activated in the right combination to become pathologic. In and of themselves, these genes are not pathologic until there is another trigger (an epigenetic factor). Remember, the FQ’s are very metabolically intense, with the ability to affect millions of points within the human body. It is what makes them both effective and deadly at the same time. This is why their adverse effects can appear long after cessation of the drug.
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I know we get tired of petitions to the point of complacency. However, please, have everyone you know sign this petition. Those who reap obscene profits, not by normal healthy capitalistic ways, but instead by corrupt influence of governmental regulation need to be held accountable. They are the reason why some of these drugs stay on the market despite serious health concerns.

www.change.org/p/u-s-department-of-justice-do-you-need-financial-help-due-to-a-medication-side-ef...
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...damaged by fluoroquinolones in 2007 at age 46. Prior to, a healthy law enforcement official. Now an amateur FQ researcher, author, and blogger.